Abstract
IT has been reported earlier that stressor agents like epinephrine1 and adrenocorticotrophic hormone2 cause a loss of alkaline phosphatase activity from the adrenal cortex. Administration of serum gonadotrophin to epinephrine-treated pigeons partially prevents this enzymatic loss1. The present communication records an attempt to determine whether a comparable reduction in alkaline phosphatase activity occurs in the adrenal cortex in response to a bacterial infection stress.
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 51 print issues and online access
$199.00 per year
only $3.90 per issue
Buy this article
- Purchase on SpringerLink
- Instant access to the full article PDF.
USD 39.95
Prices may be subject to local taxes which are calculated during checkout
Similar content being viewed by others
References
Kar, A. B., Proc. Nat. Inst. Sci. Ind., 17, 357 (1951).
Kar, A. B., Proc. Nat. Inst. Sci. Ind., 19, 487 (1953).
Gomori, G., J. Cell. and Comp. Physiol., 17, 71 (1941).
Li, C. H., and Evans, H. M., “Recent Progress in Hormone Research”, 3, 3 (Academic Press, Inc., New York, 1948).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
KAR, A. Effect of Experimental Tuberculosis on the Distribution of Alkaline Phosphatase in the Adrenal Cortex of the Guinea Pig. Nature 173, 210 (1954). https://doi.org/10.1038/173210a0
Issue date:
DOI: https://doi.org/10.1038/173210a0


