Table 1 Age and sex of the patients, causative factors for the development of disease, histopathological diagnoses and TNM classification

From: Inherent growth advantage of (pre)malignant hepatocytes associated with nuclear translocation of pro-transforming growth factor α

Age

Sex

Aetiology

Liver changes

Diagnosis

27

f

Glycogenosis 1

S, glycogenosis

HCA

33

f

 

S

HCA

46

f

  

HCA

39

m

 

S, H

DN-LG

37

f

 

S, H

DN-LG

48

m

 

F

DN-LG

25

f

 

U

DN-LG

52

f

 

U

DN-HG

55

m

HCV

C

DN-HG

61

m

HCV

C

DN-HG

50

m

HCV

C

DN-HG

77

m

 

C

DN-HG

63

f

  

HCC/1, pT1, pN0, pM0

59

f

  

HCC/1, pT2, pN0, pM0

67

m

 

C

HCC/2, pT2, pN0, pM0

64

m

Ethanol

C

HCC/2, pT3, pN0, pM0

68

m

  

HCC/2, pT3, pN0, pM0

59

f

HCV

C

HCC/2, pT3, pNx, pM0

53

m

Ethanol

C

HCC/2, pT4, pN0, pM0

63

m

HCV

 

HCC/2, pT4, pN0, pM0

58

m

Ethanol

C

HCC/3, pT4, pNx, pM0

77

f

  

HCC/3, pT4, pNx, pMx

  1. m, male; f, female; HCV, hepatitis-C-virus infection; C, cirrhosis, F, fibrosis, H, hepatitis; S, steatosis; U, unaltered liver parenchyma; HCA, hepatocellular adenoma; DN-LG, low-grade dysplastic nodule; DN-HG, high-grade dysplastic nodule; HCC, hepatocellular carcinoma. HCC/1–3 refers to the histopathological grading of the HCCs according to published criteria (Edmondson and Steiner, 1954). The TNM staging (tumour/node/metastasis) of the disease followed the UICC guidelines (Hermank et al, 1993).
Back to article page