Figure 4

KN-93 exerts an antiarrhythmic effect on CPVT-iPSC-derived CMs. Representative traces of evoked (a) and spontaneous (b) APs from CPVT-iPSC in the presence of a β-adrenergic stimulus (1 μM Iso). DADs are indicated by arrows. (c) Coperfusion with 1 μM KN-93, a CaMKII inhibitor, prevented this arrhythmogenic effect in CPVT-iPSC-derived CMs (n=7, P<0.05)