Figure 6
From: MiR-216a: a link between endothelial dysfunction and autophagy
miR-216a inhibition reduces ox-LDL accumulation and monocytes adhesion on PDLs44 HUVECs. (a) Endogenous miR-216a expression in PDLs44 HUVECs treated with ox-LDL (100 μg/ml, 16 h) (*P<0.03 Student’s t-test, data are mean ± S.E.M.). PDLs44 HUVECs transfected with A216a and treated with ox-LDL or Dil-ox-LDL were analyzed for: (b) LC3B immunofluorescence ( ***P<0.0005, one-way ANOVA. Data are mean ± S.E.M.), (c) Dil-ox-LDL fluorescence (***P<0.0001 Student’s t test, data are mean ± S.E.M.), d, THP-1 monocyte adhesion. (*P<0.05, one-way ANOVA. Data are mean ± S.E.M.). (n=4 for each experiment)
