Figure 6

Rg5 ameliorated mitochondrial dysfunction in cardiomyocytes. Neonatal rat ventricular myocytes (NRVMs) were stimulated with palmitate (PA, 100 μM) for 2 h. (a) Intracellular ROS production was viewed by dihydroethidium staining labeling with confocal microscopy. Scale bar, 100 μm; (b) Intracellular calcium concentration. *P<0.05 versus PA treatment; #P<0.05 versus indicated treatment; Data are expressed as mean±S.D. (n=4). (c) Calcein-AM combined with CoCl2 were used in viewing mitochondrial transition pore opening in cardiomyocytes. Scale bar, 10 μm. (d) Mitochondrial potential (Δψm) was viewed by Tetramethylrhodamine ethyl ester perchlorate (TMRE) labeling with confocal microscopy. Scale bar, 10 μm. The results are from three independent experiments