Figure 4: Components of the dynein-2 complex are co-expressed in a human cartilage growth plate.
From: Mutations in DYNC2LI1 disrupt cilia function and cause short rib polydactyly syndrome
(a) Sections from a 14-week human humerus were stained for DYNC2LI1, DYNC2H1 and WDR34. All three proteins were expressed in the perichondrium/periosteum and primary spongiosa (closed arrows, perichondrium/periosteum; open arrows, primary spongiosa). Scale bar, 50 μm.
