Supplementary Figure 5: Epidermal activation of AhR by air pollutants causes AD-like phenotypes through artemin expression and hyper-innervation into the epidermis. | Nature Immunology

Supplementary Figure 5: Epidermal activation of AhR by air pollutants causes AD-like phenotypes through artemin expression and hyper-innervation into the epidermis.

From: The aryl hydrocarbon receptor AhR links atopic dermatitis and air pollution via induction of the neurotrophic factor artemin

Supplementary Figure 5

Schema of AhR-mediated development and the exacerbation of AD. Note that AhR is activated by air pollutants, such as traffic-derived particulate matter. Activated AhR induces the expression of TSLP, IL-33 and ARTEMIN in keratinocytes. ARTEMIN induces hyper-innervation of the epidermis by sensory nerves, resulting in alloknesis. Subsequent scratching behaviors lead to barrier damage and increased antigen penetration, resulting in enhanced sensitization that promotes a predisposition to other allergic diseases.

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