Figure 7

Schematic representation of the miR-206 proposed mode of action in modulating PDAC tumor responses. miR-206 functions as a tumor suppressor in PDAC cells by directly dampening the antiapoptotic KRAS-induced NF-κB pathway activity, thus leading to transcriptional inhibition of the expression of multiple pro-angiogenic, pro-lymphangiogenic and growth factors, thus limiting endothelial (EC) and lymphatic endothelial cell activation and tumor growth. In addition, miR-206 inhibits ECM remodeling by directly targeting ANXA2, which serves as a proteolytic center for ECM degradation.