Abstract
Previous studies have shown lactose malabsorption to be a frequent occurrence in the premature infant. We analyzed breath hydrogen in two sets of identical prematurely born twins of 30 & 34 weeks gestation. Except for perinatal asphyxia in one infant all had uncomplicated antenatal and postnatal courses. The lactose intake was similar for each infant (g/kg).
Breath hydrogen determinations (PPMH2) are listed below:
The only asphyxiated infant (IIB) had higher peak breath hydrogen levels than its own twin (IIA) or twins I A & B. These elevated values are associated with lactose malabsorption.
Although the number of subjects in the study is small, the two sets of identical twins offer a unique opportunity to study the effects of asphyxia on lactose malabsorption in premature infants. These data in identical twins support our previous preliminary findings of lactose malabsorption associated with asphyxia in the newborn. The findings may have implications on the initial choice of feeding in asphyxiated premature infants.
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Katzenstein, M., Berezin, S., Newman, L. et al. ASPHYXIA AS A CAUSE OF LACTOSE MALABSORPTION IN IDENTICAL PREMATURE TWINS. Pediatr Res 18 (Suppl 4), 329 (1984). https://doi.org/10.1203/00006450-198404001-01418
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DOI: https://doi.org/10.1203/00006450-198404001-01418
