Fig. 7: Schematic illustration showing the possible mechanisms by which OGT modulates FAM134B-mediated ER-phagy and promotes cell survival.

Under ND conditions, FAM134B is possibly O-GlcNAcylated by OGT, which suppresses its ubiquitination-mediated degradation. Subsequently, enhanced FAM134B-mediated ER-phagy restores intracellular homeostasis and facilitates cell survival.