Fig. 5: Schematic representation of the potential mechanism of CN tumorigenesis.

The middle left and upper panels show the normal course of radial glial cell differentiation into mature neurons. The middle right and lower panels show the potential course of CN tumorigenesis, whereby FGFR3 hypomethylation-driven FGFR3-overexpressing radial glial-like cells progress through the ventricle as a result of PIK3-AKT activation and altered neuronal functions such as differentiation and migration.