Fig. 5: Comparison of the mutational profiles of 68 GCAs in this study with those reported in usual endocervical adenocarcinomas, pancreatic adenocarcinomas, and intestinal/tubular gastric adenocarcinomas. | Modern Pathology

Fig. 5: Comparison of the mutational profiles of 68 GCAs in this study with those reported in usual endocervical adenocarcinomas, pancreatic adenocarcinomas, and intestinal/tubular gastric adenocarcinomas.

From: Massively parallel sequencing analysis of 68 gastric-type cervical adenocarcinomas reveals mutations in cell cycle-related genes and potentially targetable mutations

Fig. 5

Comparison of somatic non-synonymous mutations identified in GCAs in the present study (n = 68) with those of HPV-associated usual endocervical adenocarcinomas (UEAs), pancreatic adenocarcinomas (PAs) and intestinal gastric adenocarcinomas (IGAs), as reported in The Cancer Genome Atlas (TCGA), retrieved from cBioPortal [37, 38]. Fisher’s exact test, *p < 0.05; **p < 0.01. Indel, small insertion and deletion, SNV single nucleotide variant.

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