Fig. 6 | Mucosal Immunology

Fig. 6

From: Helicobacter suis infection alters glycosylation and decreases the pathogen growth inhibiting effect and binding avidity of gastric mucins

Fig. 6

H. suis growth in the presence of gastric mucins from infected or noninfected pigs and humans. a, b Growth of H. suis in the presence of pig gastric mucins isolated from H. suis infected or noninfected pigs. The results are presented in relation to growth in the absence of mucins. a H. suis growth in response to four mucin sources per individual (i.e., surface soluble, surface insoluble, gland soluble, and gland insoluble mucins). Statistics: ***p < 0.001, two-way ANOVA, the bars represent mean ± SEM. b Mean H. suis growth in response to the four mucin sources. The graph shows one data point per individual. Statistics: *p ≤ 0.05, Mann–Whitney U test, the bars represent median with range. c H. suis growth in the presence of human gastric mucins isolated from H. pylori-infected or noninfected individuals. The results are presented in relation to H. suis cultured without mucins. Statistics: *p < 0.05, Mann–Whitney U test, the bars represent median with range. The graph shows one data point per individual. Surface soluble, Gland soluble, □ Surface insoluble, ■ Gland insoluble, ▲ Mean growth in response to the four mucin sources per pig. The pig marked in gray carried a second H. suis strain in addition to the strain used in the experimental infection. The graphs are representative of two experiments

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