Table 3 Relationship between cardiac concentricity and non-hypertensive pathway measures in schizophrenia.

From: Adipose tissue dysfunction, inflammation, and insulin resistance: alternative pathways to cardiac remodelling in schizophrenia. A multimodal, case–control study

 

Whole sample

Schizophrenia sample

Supplementary Figs.

R2

F (48, 1)

β for concentricity

p

BH p

R2

F (24,1)

β for concentricity

p

BH p

Log(adiponectin)

0.20

11.70

−1.16

0.001

0.01

0.15

4.12

−0.91

0.05

0.378

2

Log(triglycerides)

0.11

6.05

1.07

0.02

0.06

0.07

1.77

0.82

0.20

0.392

3

Log(endothelin-1)

0.10

5.12

0.50

0.03

0.06

0.05

1.38

0.39

0.25

0.392

4

Log(ALP)

0.02

1.22

0.48

0.27

0.38

0.03

0.67

0.26

0.42

0.490

5

Log(HDL)

0.06

3.30

−0.40

0.08

0.13

0.05

1.39

−0.31

0.25

0.392

6

Log(hsCRP)

<0.01

0.08

0.30

0.78

0.78

<0.01

0.06

0.25

0.81

0.810

7

Log(fasting glucose)

0.01

0.60

0.07

0.44

0.51

0.05

1.24

−0.13

0.28

0.392

8

  1. Simple linear regression analysis was used to test the association between significantly altered non-hypertensive pathway measures and participants’ concentricity values.
  2. ALP alkaline phosphatase, hsCRP high-sensitivity C-reactive protein, HDL high-density lipoprotein, BH Benjamini and Hochberg.
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