Table 1 APL patients’ characteristics and results of ATO therapy

From: Arsenic trioxide promoting ETosis in acute promyelocytic leukemia through mTOR-regulated autophagy

No.

Sex/age

Diagnosis

Blasts (BM%)

WBC (×109/l)

Plts (×109/l)

Hb (g/l)

Outcome

1

F/44

M3/bcr1

80

5.1

43

62

HCR

2

F/45

M3/bcr1

84

10.4

27

57

HCR

3

M/34

M3/bcr3

78

4.5

37

77

HCR

4

M/38

M3/bcr1

88

11.2

33

68

HCR

5

F/17

M3/bcr3

88

28.2

26

90

HCR

6

F/44

M3/bcr1

85

18.2

45

67

HCR

7

F/42

M3/bcr1

95

25.7

31

77

PR

8

M/41

M3/bcr1

92

26.1

22

60

PR

9

M/45

M3/bcr1

87

14.2

14

77

HCR

10

F/34

M3/bcr1

90

32.5

12

79

HCR

11

M/53

M3/bcr3

92

20.4

77

58

PR

12

F/29

M3/bcr1

91

24.4

34

86

HCR

13

M/39

M3/bcr3

94

6.2

37

66

HCR

14

M/35

M3/bcr3

94

26.2

33

76

HCR

15

M/62

M3/bcr1

80

4.5

55

76

HCR

16

M/23

M3/bcr1

89

4.1

12

62

HCR

17

M/33

M3/bcr1

82

20.2

13

61

ED

18

M/23

M3/bcr3

81

18.9

19

62

HCR

19

F/39

M3/bcr3

83

17.7

12

68

HCR

20

F/41

M3/bcr1

87

12.8

32

55

PR

Ref. range

 

0–0.4

4–10

100–300

110–170

HCR

  1. The main clinical and laboratory features of 20 newly diagnosed APL patients and results of ATO therapy during induction therapy
  2. Blasts promyelocytes + blasts, PB peripheral blood, WBC white blood cell, Plts platelets, Hb hemoglobin, bcr breakpoint cluster region (bcr1 = intron 6, bcr3 = intron 3), HCR complete hematologic remission, PR partial remission, ED early death