Table 1 Characteristics of patients with biliary atresia (BA) or choledochal cysts (CCs)

From: MicroRNA-29b/142-5p contribute to the pathogenesis of biliary atresia by regulating the IFN-γ gene

 

BA

CCs

p-value

Age (months)

2.1 ± 0.6

18.5 ± 3.2

>0.05

Male

35

11

N/A

Female

25

9

N/A

Type

IIIa

Ib

N/A

ALP (IU/L)

625.5 ± 28.7

77.6 ± 12.4

<0.05

ALT (IU/L)

122.6 ± 20.2

12.6 ± 5.6

<0.05

AST (IU/L)

176.4 ± 22.4

18.3 ± 7.2

<0.05

DBIL (umol/L)

121.1 ± 6.2

3.2 ± 1.2

<0.05

TBIL (umol/L)

145.1 ± 12.2

7.8 ± 2.3

<0.05

GGT (IU/L)

686.8 ± 86.4

36.4 ± 13.6

<0.05

TBA (umol/L)

126.2 ± 8.2

7.5 ± 2.2

<0.05

  1. aType III atresia refers to the discontinuity of both right and left hepatic ducts to the level of the porta hepatis
  2. bType I is the most common (80–90%) involving saccular or fusiform dilatation of a portion or entire common bile duct with normal intrahepatic duct. CCs patients without jaudice have normal liver function as controls
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