Fig. 8: Effect of HPC on the activity of Na+/K+-ATPase in CA1 after tGCI. | Cell Death & Disease

Fig. 8: Effect of HPC on the activity of Na+/K+-ATPase in CA1 after tGCI.

From: Neuroglobin mediates neuroprotection of hypoxic postconditioning against transient global cerebral ischemia in rats through preserving the activity of Na+/K+ ATPases

Fig. 8

A Quantitative analysis of the NKA activity in the CA1 subregion in Sham, tGCI, and HPC groups. B Quantitative analysis of the NKA activity in the Sham and the HPC groups with or without ODNs administration. C Quantitative analysis of the NKA activity in the Sham and the tGCI groups with or without lentiviral administration. Data are expressed as percentage of the value of Sham-operated animals. Each bar represents the mean ± S.D. *p < 0.05 vs. Sham-operated animals, #p < 0.05 vs. tGCI group, &p < 0.05 vs. HPC group with injection of S-ODNs, and △p < 0.05 vs. tGCI group with injection of Lenti-control (n ≥ 4 in each group)

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