Fig. 7: Effects and mechanisms of TMEM16A on LPS-induced IEC-6 cell apoptosis. | Cell Death & Disease

Fig. 7: Effects and mechanisms of TMEM16A on LPS-induced IEC-6 cell apoptosis.

From: Dual role of Ca2+-activated Cl− channel transmembrane member 16A in lipopolysaccharide-induced intestinal epithelial barrier dysfunction in vitro

Fig. 7

Cells were cultured for 24 h with media contained siRNA-targeting TMEM16A (TMEM16A–) or adenovirus stimulating TMEM16A (TMEM16A+) in the presence and absence of high-dose LPS. a Effects of TMEM16A- and TMEM16A + on high-dose (10 μg/mL) LPS induced corresponding protein expressions in IEC-6 cells. (n = 3/group). b Effects of TMEM16A– and TMEM16A+ on high-dose (10 μg/mL) LPS induced corresponding protein expressions in IEC-6 cells. (n = 3/group). Data are expressed as the mean ± SD. Values in the normal (NC) group are set to 100% and other values are given relative to those in the NC group, **p < 0.01 compared with corresponding control (same color) group or as indicated.

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