Fig. 2: miR-103a expression was upregulated in HCC and related to poor prognosis in the Renji cohort. | Cell Death & Disease

Fig. 2: miR-103a expression was upregulated in HCC and related to poor prognosis in the Renji cohort.

From: MiR-103a promotes tumour growth and influences glucose metabolism in hepatocellular carcinoma

Fig. 2

A Reverse transcription-quantitative PCR analysis was performed to assess miR-103a expression in 89 paired HCC tissues and adjacent normal tissues and two healthy volunteer liver tissues. B Survival curves suggested that increased miR-103a expression was significantly associated with unfavourable prognosis of patients with HCC. CF Reverse transcription-quantitative PCR analysis was performed to validate miR-103a expression in SMMC-7721 (p < 0.001), MHCC97H (p < 0.001), MHCC97L (p < 0.01) and HCCLM3 (p < 0.001) cells transfected with miR-103a lentivirus. All results are representative of at least three independent experiments.

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