Fig. 6: Effects of bone marrow mesenchymal stem cell-derived extracellular vesicles on renal fibrosis by regulating miR-181d/KLF6/NF-κB axis.

Note: A diagram showing the mechanism whereby miR-181d is transferred by BMSC-EVs into neighboring cells where it targets KLF6, thereby inactivating the NF-κB signaling pathway and alleviating renal fibrosis in rats.