Fig. 5: SLC1A5 resisted cellular oxidative stress damage and ferroptosis state in glioma cells. | Cell Death & Disease

Fig. 5: SLC1A5 resisted cellular oxidative stress damage and ferroptosis state in glioma cells.

From: SLC1A5 enhances malignant phenotypes through modulating ferroptosis status and immune microenvironment in glioma

Fig. 5

A The CCK-8 assay results of cell viability after SLC1A5 knockdown with Erastin treatment. B The MDA level after SLC1A5 knockdown with Erastin treatment. C The level of reduced GSH after SLC1A5 knockdown with Erastin treatment. D The fluorescence intensity of DHFC-DA (green) in SLC1A5 knockdown T98G cells with Erastin treatment (Scale Bar = 20 μm). E The level of oxidized lipid peroxides(green) in SLC1A5 knockdown T98G cells with Erastin treatment (Scale Bar = 20 μm). F The HE staining and the tumor size analysis in Erastin and V9302 separate and combined treatment groups (Scale Bar = 1 mm). G Kaplan–Meier survival curves of survival time in different treatment groups. H The Ki-67 IHC images and scores of glioma sections in different treatment groups (Scale Bar = 20 μm).

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