Fig. 7: Schematic representation of the functional role of HNF1A-AS1 in regulating GCSCs.
From: CMYC-initiated HNF1A-AS1 overexpression maintains the stemness of gastric cancer cells
HNF1A-AS1 together with CMYC and miR-150-5p formed a positive feedback loop to activate β-catenin expression in GC.
