Fig. 7: Proposed model of the action of galectin-8 in CRC.

CRC cells showed increased levels of B4GALT1. In the absence of galectin-8 (left), TGF-β binds to TβRII, thereby promoting EMT. In the presence of galectin-8 (right), galectin-8 binds to TβRII through B4GALT1-mediated galactosylation of N-glycans, resulting in a decrease of TGF-β signaling-mediated EMT.