Fig. 2: The function of ferroptosis in H. pylori infection gastric cancer.

H. pylori infection could be up-regulated or downregulated in gastric mucosal cells. H. pylori mediated the expression of TCF4 and then regulated GPX4 and ROS, influenced cell ferroptosis. Also, H. pylori and its infection-regulated PHKG2 expression mediated ALOXs influenced ferroptois. Furthermore, the OMVs and CagA all play an important role in the regulation of ferroptosis mediated by H. pylori.