Fig. 2: MAF proteins involvement in hematological diseases.
From: MAFB: a key regulator of myeloid commitment involved in hematological diseases

A MAFB overexpression in multiple myeloma characterized by the t(14;20)(q32;q11) translocation. B Generation of a transgenic mouse to study the role of c-Maf in T cell lymphoma genesis and comparison with c-MAF expression levels in patients with angioimmunoblastic T cell lymphoma (AITCL). C Overexpression of MAFB in acute monoblastic leukemias characterized by DNMT3A R882 mutation.