Fig. 2
From: NKG2A is a NK cell exhaustion checkpoint for HCV persistence
Impaired hepatic NK cell response during HCV persistent infection in C/OTg mice. Mice were treated as in Fig. 1a. FACS analyses of intrahepatic NK cells for expression of a DX5, b CD11b, and CD27, c activation receptors, including Ly49D, Ly49H, and NKG2D, and d inhibitory receptors, including NKG2A, KLRG1, and TIGIT. e FACS analyses of peripheral NK cells for expression of NKG2A and KLRG1. f NK function assays were carried out at indicated time of HCV infection by Yac-1 cell stimulation. CD107a and IFN-γ expression were FACS analyzed. Data were mean ± SD, ANOVA test. *P < 0.05; **P < 0.01. Source data are provided as a Source Data file
