Fig. 10
Proposed mechanism for the mitochondria-specific self-circulation of CPT release and mtROS burst to damage mitochondria and initiate cell apoptosis/death by dual-targeted polyprodrug nanoreactors (DT-PNs)
Fig. 10 | Nature Communications
Proposed mechanism for the mitochondria-specific self-circulation of CPT release and mtROS burst to damage mitochondria and initiate cell apoptosis/death by dual-targeted polyprodrug nanoreactors (DT-PNs)
