Fig. 8: RAS co-opts mTORC1 and the amino acid sensing machinery to drive oncogenic growth in MM.

Model of oncogenic RAS signaling in MM: (1) Oncogenic isoforms of RAS (KRAS or NRAS) promote localization of SLC3A2 and MTOR to endolysosomes with oncogenic RAS. (2) RAS associates with PI3-K and TSC2 to promote full activation of mTORC1 signaling. (3) RAS stimulates classical MAPK signaling.