Fig. 5: Prophylactic blockade of PGE2 signaling through the EP2 receptor improves survival to IAV infection in aged mice.

a, b Aged (18â22 months) female C57BL/6 mice were given 7 daily i.p. injections of the EP2 antagonist starting 7 days before infection or 11 daily i.p. injections of the EP2 and EP4 antagonists starting 7 days before infection. The mice were then infected with 400âpfu of PR8 IAV intranasally as depicted in (a). Survival of mice (b) was tracked daily. nâ=â14 for Veh Control, nâ=â6 for EP2 Antg, and nâ=â9 for EP2â+âEP4 Antg. c, d Aged mice were given clodronate-loaded liposomes followed by an adoptive transfer of WT or EP2 KO AMs. 4 weeks following the adoptive transfer, the mice were given EP2 antagonist or vehicle control for 7 days prior to infection with 400âpfu of PR8 IAV intranasally as depicted in (c). Survival of mice (d) was tracked daily. nâ=â8 for WT AMs+EP2 Antg. nâ=â9 for EP2 KO AMs+ Veh Control and WT AMs + Veh Control. nâ=â10 for EP2 KO AMs+ EP2 Antg. For panels b and d, survival differences were statistically determined by two-tailed GehanâBreslowâWilcoxon test. Schematics created in BioRender. Source data are provided as a Source Data file.