Correction to: Nature Communications https://doi.org/10.1038/s41467-020-19227-5, published online 23 October 2020
The original version of this article contained an error in the labelling of the bar graphs to the right of Fig. 3b, 3c, 3d and 3e, in which the symbols + and – representing addition or absence of the SYK inhibitor R406, respectively, were inadvertently switched. This has been corrected in the PDF and HTML versions of the article.
The original version of this article also contained an error in the title of the legend for Fig. 2. which incorrectly read ‘TREM2 knockout microglia exhibit decreased caspase activation at baseline and after cytokine starvation’. The correct version states ‘increased’ in place of ‘decreased’. This has been corrected in both the PDF and HTML versions of the article.
Author information
Authors and Affiliations
Corresponding author
Additional information
The original article can be found online at https://doi.org/10.1038/s41467-020-19227-5.
Rights and permissions
Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.
About this article
Cite this article
McQuade, A., Kang, Y.J., Hasselmann, J. et al. Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease. Nat Commun 14, 1194 (2023). https://doi.org/10.1038/s41467-023-36930-1
Published:
DOI: https://doi.org/10.1038/s41467-023-36930-1