Fig. 8: Atypical ALK fusions show a poor survival compared to typical ALK fusions.
From: Mechanistic patterns and clinical implications of oncogenic tyrosine kinase fusions in human cancers

a, b Progression-free (a) and overall (b) survival in patients carrying typical and atypical ALK fusions treated with ALK inhibitors. P-values were determined by two-sided, and confidence intervals were at the 95% level. c OncoPrint plot showing the top 42 genes mutated among NSCLC harboring typical and atypical ALK fusions. TMB tumor mutational burden, SNVs single nucleotide variants.