Fig. 10: Proposed model for GmERF13-mediated salt stress inhibition of nodulation. | Nature Communications

Fig. 10: Proposed model for GmERF13-mediated salt stress inhibition of nodulation.

From: GmERF13 mediates salt inhibition of nodulation through interacting with GmLBD16a in soybean

Fig. 10

Under normal conditions, rhizobia activated by root flavonoids release NFs to stimulate the symbiotic signaling pathway. The downstream factor GmLBD16a regulated by GmNINs is required for the expression of the symbiotic gene GmEXP17c by directly binding to its promoter, thereby promoting the production of nodules. Under salt stress conditions, key transcriptional factors such as GmABI5a and GmABI5b in the ABA classic signaling pathway directly activate the expression of GmERF13s. The upregulated GmERF13s subsequently interact with GmLBD16a, thereby reducing its binding ability to the downstream gene GmEXP17c’s promoter and causing a significant reduction in the number of nodules.

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