Fig. 3
From: Genetic interaction networks mediate individual statin drug response in Saccharomyces cerevisiae
Hierarchical clustering of GINs represents overall genetic background variation throughout the genome and specifically the unfolded protein response and mevalonate pathway. a–d Hierarchical clustering of growth phenotypes showing a genome-wide deletion libraries, b IRE1 (the major mediator of UPR), c HMG1 and HMG2 (the targets of atorvastatin and cerivastatin), and d genes within and branching from the mevalonate pathway. One scale applies to a–c and the scale for d shown separately to the right of the cluster. All scales represent min–max normalised Z-scores
