Extended Data Fig. 6: Characterization of the role of Ubx2 in CCCP-induced precursor accumulation.
From: Mitochondrial protein translocation-associated degradation
a, b, Wild-type, ubx2∆, ubx2∆ + UBX2 (a) and shp1∆ (b) cells were either left untreated or treated with CCCP for the indicated time periods. Cell extracts were analysed by SDS–PAGE and immunodetection. The data are representative of three independent experiments.
