Extended Data Fig. 8: Commensal bacteria influence CNMa expression in the gut, but not in the fat body or brain.
From: Response of the microbiome–gut–brain axis in Drosophila to amino acid deficit
a, Quantifications of GFP fluorescence in the R2 region of the gut of conventionally reared and GF female flies carrying CNMa-Gal4>UAS-GFP fed on 10% or 15% dietary yeast. b, c, The level of CNMa expression in the fat body (b) or the brain (c) of flies bearing CNMa-Gal4 and UAS-GFP that had been reared in 10% dietary yeast were quantified using the GFP signal intensity measured by its native GFP fluorescence. d, Quantifications of GFP fluorescence in the R2 region of GF flies that had been mono-associated with A. pomorum or L. plantarum WJL. Data are mean ± s.e.m. P values are indicated; one-way ANOVA with Tukey’s post-hoc test in a, d; unpaired two-tailed t-test in b, c. Sample sizes and statistical analyses are shown in Supplementary Table 1.
