Fig. 3: Arterial occlusion-induced peripheral pulselessness is similar to central pulselessness from VF, as measured at the wrist using PPG.
From: Automated loss of pulse detection on a consumer smartwatch
a, Central pulselessness from VF induced in an electrophysiology laboratory is of short duration (<10 s) and contains artefacts from shocking the heart into and out of the arrhythmia. a.u., arbitrary units. b, Peripheral pulselessness induced through arterial occlusion is of longer duration (>60 s). c,d, Spectrograms of the central (c) and peripheral pulselessness (d) windows are similar and show the absence of pulsatility at typical pulse rates, 40–220 BPM. e, There is no significant difference between the maximum power at these frequencies during central pulselessness and peripheral pulselessness, as assessed by comparing the medians across both groups (P = 0.985, Mann–Whitney U-test, two-sided). NS, not significant. The box plot shows the median, quartiles and the range of the non-outlier data points, overlaid with all individual data points.
