Figure 7 | Scientific Reports

Figure 7

From: HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entry

Figure 7

HPV-16 PsVs in senescent cells are accessible to neutralising antibody. (a) Anti-L1 (H16.V5) neutralising antibody binds to the surface of senescent and proliferating cells. Senescent BJ cells were transfected with control siRNA and p53 siRNA. 48 h after transfection cells were incubated with anti-L1 neutralising antibody for 24 h. Then cells were fixed and neutralising antibody was detected with goat anti-mouse rhodamine antibody (red) and rabbit antibody against EEA-1, and detected using Alexa Fluor 647-conjugated donkey anti-rabbit (blue). Two examples are shown for each condition. (b) In infected senescent cells the H16.V5 neutralising antibody co-localises with the virus on the surface of cells. One day after siRNA transfection, AF488-labelled HPV-16 PsVs (green) were added to the cells. 24 h after infection cells were incubated overnight with neutralising H16.V5 antibody, fixed and stained as above. Note that in senescent cells transfected with the control siRNA, H16.V5 neutralising antibody co-localises with the virus on the cell surface (indicated by arrows). In cells that were transfected with the p53 siRNA there is very little co-localisation between the virus and neutralising antibody and virus is visible within the cells, as indicated by the arrows, suggesting that virus entry is stimulated post–transfection of the p53 siRNA.

Back to article page