Figure 7
From: The schizophrenia risk locus in SLC39A8 alters brain metal transport and plasma glycosylation
Plasma protein N-glycan changes in severe loss-of-function SLC39A8 mutation carriers after Mn treatment. Data presented as percent abundance of (a) tri-antennary, (b) tetra-antennary, (c) high-mannose, and (d) bisecting N-glycans before and after ~ 1 year of Mn supplementation in subjects A and B with congenital disorders of glycosylation due to severe SLC39A8 homozygous mutations. Samples from each individual were replicated twice with similar results.
