Figure 8

Putative models of host genotype-specific symbiotic interactions between bradyrhizobia and V. radiata varieties may be controlled by type III effector NopEs. Hypothetically, NopEs are nodulation determinants capable of providing the ability to nodulate V. radiata varieties, including KPS1, possibly by modulating downstream symbiosis signalling and/or suppressing the phytohormone-mediated effector-triggered immunity type (ETI-type) defence response (PmETI type). On the other hand, NopEs might be recognized by unknown receptor(s)/a specific resistance (R) protein in the KPS2 variety, consequently activating an R protein-mediated ETI-type defence response (RmETI-type) restricting nodulation. The dotted line presents the unclear symbiotic mechanism.