Fig. 2 | Scientific Reports

Fig. 2

From: Prenatal bisphenol A exposure causes sperm quality and functional defects via Leydig cell impairment and meiosis arrest in mice offspring

Fig. 2

Morphology and histology of the male genital tracts of prenatal BPA-exposed mice offspring.Morphology of the male genital tracts of control and prenatal BPA-exposed mice offspring (A). T (testis), SV (seminal vesicle), VD (vas deferens), EP (epididymis). Litter numbers of F1 generation in control and BPA-exposed pregnant mice. Characteristics of body weight, testis weight, testicular index, sperm count and daily sperm production in control and prenatal BPA-exposed offspring. (B) Histopathology of testes of control and prenatal BPA-exposed offspring. The sections were demonstrated by HE staining. Sp (spermatogonia), Ps (spermatocyte), Rs (round spermatid), Es (elongated spermatids), Lc (leydig cells). Percentages of testicular stage VII and VIII tubules. The number of Leydig cells in interstitial space in control and prenatal BPA-exposed offspring. Testosterone concentration in control and prenatal BPA-exposed offspring. Each bar represented 100 μm. The data were analyzed by t-test, p < 0.05 was considered significance. Each bar represented 100 μm.

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