Abstract
Selective pressure from antimicrobial use can drive the emergence of methicillin-resistant Staphylococcus aureus (MRSA) isolates. We evaluated resistance rates to fluoroquinolones and chlorhexidine digluconate (CD) and the effect of their selective pressure on lineages of MRSA. Minimum inhibitory concentrations (MICs) for fluoroquinolones and CD were determined for 75 isolates from 11 lineages. Phenotypic expression of efflux pumps was assessed by the Cartwheel method and associated genes were detected by PCR. Cross-resistance between antibiotics and CD and point gene mutations were investigated after exposure to increasing concentrations of fluoroquinolones and CD. The MIC90 values for ciprofloxacin, moxifloxacin, and CD in 75 MRSA isolates were 128, 8 and 1 mg/L, respectively. ST5-SCCmecII, ST239-III and ST1-IV lineages showed the highest resistance rates to fluoroquinolones, and ST5-II showed the highest MIC90 for CD. Among 17 (22.6%) MRSA isolates with detectable efflux pump activity, seven showed a higher efflux potential, including four ST5-II isolates. The qacA/B genes were found in 14 (18.6%) isolates, and 10 were ST5-II, while the smr gene was frequent among isolates with SCCmecIV (71.4%). Eight of 10 selected strains showed increased resistance to fluoroquinolones and/or tetracycline and increased phenotypic expression of efflux pumps. Point mutations in the gyrA, parC, norA, and/or norB genes emerged in three strains after exposure to higher doses of ciprofloxacin or CD. Resistance to fluoroquinolones was high among MRSA isolates, especially those from ST5-II. MRSA isolates subjected to in vitro selective pressure of antimicrobials showed increased MICs and gene mutations, reinforcing the importance of the rational use of antimicrobial agents in clinical practice.
Data availability
The data sets generated and analyzed during the current study, such as the patterns of genetic mutations identified after induction with ciprofloxacin and CD, are available in Genbank (Access number: PX432114 → PX432119).
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Acknowledgements
We thank FIOCRUZ-INCQS, Rio de Janeiro, RJ for the donation of Staphylococcus aureus INCQS 00039 (ATCC 6538) strain of Collection of Reference Microorganisms in Health Surveillance-CRMVS. We also thank Dr. Renata Cristina Picão/UFRJ for the donation of P. aeruginosa PA01XY++ strain.
Funding
This study was financed in part by the Coordenação de Aperfeiçoamento de Pessoal de Nível Superior - Brasil (CAPES) - Finance Code 001, Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro [FAPERJ, Grants #E-26/203.296/2017; #E-26/200.419/2023, #E-26/010.000172/2016, #E-26/010.001463/2019, #E-26/010.101056/2018; #E-26/211.554/2019 (Projeto REDES); #E-26/201.071/2020; #E-26/211.284/2021; #E-26/201.454/2021 and #E-26/205.939/2022] and Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq, Grants #307594/2021-1 [KRNS] and 405020/2023-6 [Universal-MSL]).
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TLRO and KRNS designed the study. TLRO, AFS, MGS and BMS analyzed the S. aureus isolates, conducted the experiments described in the “Methods” sections. TLRO gathered the data for the final statistical analysis as well as drafted the manuscript. RBRF and RSD critically reviewed the manuscript. KRNS supervised and critically reviewed the manuscript. All authors approved the final version.
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This study was approved by the Human Research Ethics Committee of Universidade Federal do Rio de Janeiro (UFRJ) under numbers 159/07 and CAAE40652714.0.0000.5257, and by the Human Research Ethics Committee of Copa Dor hospital under number 088/07. In addition, all methods were performed in accordance with the relevant guidelines and regulations. All experimental protocols were approved by the Microbiology Institute/UFRJ.
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de Oliveira, T.L.R., de Souza, A.F., de Souza, B.M. et al. Antimicrobial susceptibility and adaptative changes in MRSA lineages exposed to increasing concentrations of fluoroquinolones and chlorhexidine. Sci Rep (2026). https://doi.org/10.1038/s41598-026-40345-5
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DOI: https://doi.org/10.1038/s41598-026-40345-5