Table 1 Physiological data of the four groups.

From: Dietary salt with nitric oxide deficiency induces nocturnal polyuria in mice via hyperactivation of intrarenal angiotensin II-SPAK-NCC pathway

 

NSD

HSD

L-NAME

L-NAME + HSD

Body weight (g)

29.6 ± 0.68

29.8 ± 0.67

29.2 ± 0.33

29.1 ± 0.82

Food intake (g)

 Active phase

3.58 ± 0.32

3.22 ± 0.26

3.32 ± 0.40

3.50 ± 0.40

 Inactive phase

0.52 ± 0.19

0.66 ± 0.29

0.58 ± 0.18

0.74 ± 0.19

 Total

4.10 ± 0.45

3.88 ± 0.37

3.90 ± 0.27

4.24 ± 0.27

Water intake (g)

 Active phase

4.42 ± 0.22

4.30 ± 0.42

4.34 ± 0.49

4.66 ± 0.39

 Inactive phase

0.60 ± 0.12

0.72 ± 0.19

0.76 ± 0.18

0.66 ± 0.09

 Total

5.02 ± 0.28

5.02 ± 0.28

5.10 ± 0.48

5.32 ± 0.36

Systolic blood pressure (mmHg)

 Active phase

107 ± 1.33

110 ± 2.76

112 ± 4.67

119 ± 2.50*

 Inactive phase

96 ± 2.69

99 ± 4.19

103 ± 4.28

114 ± 3.60*

Serum Na (mEq/L)

149 ± 2.99

148 ± 0.93

150 ± 0.98

152 ± 1.85

Serum Cr (mg/dL)

0.13 ± 0.01

0.13 ± 0.01

0.16 ± 0.01*

0.17 ± 0.02*

  1. Body weight, food intake, water intake, systolic blood pressure, as well as serum Na and Cr levels. Data are expressed as the mean ± SEM. Statistical analysis was performed using the Tukey–Kramer method. Systolic blood pressure: l-NAME + HSD vs. NSD or HSD or l-NAME, Serum Cr: l-NAME + HSD vs. NSD, l-NAME vs. NSD.
  2. *p  <  0.05 (n  =  5 mice per group).
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