Fig. 6: miR-15a is involved in regulation of known mechanisms in AAA pathogenesis and its circulating levels are dysregulated among AAA patients.

Biofluids from multiple cohorts of AAA patients were analyzed, discovering differences in expression of miR-15a and thus revealing a biomarker with potential diagnostic implications, but also shown to be concentrated in diseased areas. miR-15a has been described to play a role in the fate of aortic smooth muscle and endothelial cells. Our studies in a mouse AAA model show that its inhibition can limit growth of experimentally induced aortic aneurysms. Created in BioRender (https://BioRender.com/pg73t6h).