Extended Data Fig. 4: Correlation scatter plots demonstrating strength of linear association between flow cytometry derived subpopulation quantification or histologically determined graft size and resultant arrhythmia burden for each PSC-CM (red) and RA-PSC-CM (purple) cell dose. | Nature Cardiovascular Research

Extended Data Fig. 4: Correlation scatter plots demonstrating strength of linear association between flow cytometry derived subpopulation quantification or histologically determined graft size and resultant arrhythmia burden for each PSC-CM (red) and RA-PSC-CM (purple) cell dose.

From: Cellular heterogeneity of pluripotent stem cell-derived cardiomyocyte grafts is mechanistically linked to treatable arrhythmias

Extended Data Fig. 4

(a) Arrhythmogenic cardiomyocytes, SIRPA+CD90−CD200+ (b) Non-arrhythmogenic cardiomyocytes, SIRPA+CD90−CD200− (c) Cardiac troponin T positive cells (d) Cardiac troponin T negative cells (e) Cardiomyocytes, SIRPA+CD90− (f) Committed ventricular cardiomyocytes, CD77+CD200− (g) Fibroblasts, CD90+ (h) Endothelial cells, CD31+CD34+ (i) Non-myocytes (j) Mesodermal progenitors, CD13+ (k) Graft size as percentage of infarct area in stained section (l) Graft size as percentage of left ventricular area in stained section. Pearson correlation reported in all panels.

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