Figure 1
TMS induces synaptic plasticity after TBI.
Immunofluorescence staining with nuclear (DAPI) and synaptic plasticity (CaMKII) markers show that TBI rats that received four-week TMS treatment had an increased expression of CaMKII as is indicated by increased fluorescent intensity, compared to TBI rats not subjected to the treatment. Increases in CaMKII fluorescent intensity were found in L2–3 (a) and L4 (b). Higher magnification images of the insets are shown in the right panels. Scale bars: 20 μm. (c) Group data shows increases in CaMKII intensity in TBI + TMS rats (n = 2), compared to TBI rats (n = 2; *P < 0.05). For each rat, three brain slices around bregma 0 were selected for staining. Data was calculated from six brain slices for each group with a field of view of 0.04 mm2.
