Figure 8: The diagram shows the Proposed Molecular mechanism of HO-1 mediated Stat3 inhibition.

Persistent actived Stat3 promotes hyperproliferation and abnormal keratin expression in the keratinocytes of psoriasis patients, treatments with Hemin/CoPP induce HO-1 expression, which enhance the expression of SHP-1 in cell plasma, this result in the inhibition of Stat3 phosphorylation and the decrease of downstream genes expression.