Table 1 Demographic data and baseline information.

From: The OPRM1 A118G polymorphism modulates the descending pain modulatory system for individual pain experience in young women with primary dysmenorrhea

 

PDM (n = 61)

Control (n = 65)

p Value

Group

Genotype

Pain

Interaction

Age, year

 AA homozygotes

22.4 (2.61)

23.2 (1.64)

0.087

0.408

NA

NA

 G allele carriers

23.0 (2.04)

24.2 (2.68)

Years of menstruating

 AA homozygotes

11.3 (2.69)

10.5 (1.49)

0.484

0.325

NA

NA

 G allele carriers

10.9 (2.60)

12.0 (3.16)

Days of one menstrual cycle

 AA homozygotes

29.4 (1.55)

29.5 (1.25)

0.546

0.760

NA

NA

 G allele carriers

29.5 (1.48)

29.5 (1.53)

Edinburgh Handedness

 AA homozygotes

82.5 (17.63)

85.6 (14.35)

0.643

0.291

NA

NA

 G allele carriers

82.0 (17.81)

77.3 (20.53)

Mnemonic PRI scores*

 AA homozygotes

34.7 (12.75)

NA

NA

0.531

0.039

0.300

 G allele carriers

35.0 (14.88)

NA

Present PRI scores *

 AA homozygotes

29.5 (12.94)

NA

 G allele carriers

33.2 (13.04)

NA

  1. Normality was tested for all assessments, and only the PRI scores in the McGill Pain Questionnaire were normality (p > 0.05). The data are presented as the means (SD). NA, not applicable.
  2. *Four subjects with primary dysmenorrhea (PDM; 3 AA homozygotes, 1 G allele carrier) did not complete the pain rating index (PRI) of the McGill Pain Questionnaire and were excluded from these calculations.