Table 4 Changes in the Glycopattern of Mitochondria Glycoproteins in Cerebral Cortex of SAD rat model.

From: Comparison of the glycopattern alterations of mitochondrial proteins in cerebral cortex between rat Alzheimer’s disease and the cerebral ischemia model

Lectins

Specificity Glycans

Ratio (SAD/SAD-C)

PSA

Fucα-1,6GlcNAc, α-D-Man, α-D-Glc

2.35

WFA

terminating in GalNAcα/β1-3/6 Gal

0.61

PTL-I

GalNAc, GalNAcα-1,3 Gal, GalNAcα-1,3Galβ-1,3/4Glc

0.22

AAL

Fucα1-6 GlcNAc(core fucose), Fucα1-3(Galβ1-4)GlcNAc

0.42

PTL-II

Gal, blood group H, T-antigen

0.61

SBA

α- or β-linked terminal GalNAc, (GalNAc)n, GalNAcα1-3 Gal

0.57

UEA-I

Fucα1-2Galβ1-4Glc(NAc)

0.53

PHA-E + L

Bisecting GlcNAc, bi-antennary N-glycans tri- and tetra-antennary complex-type N-glycan

0.34

SNA

Sia2-6Gal/GalNAc

0.62

  1. Changes were identified by lectin microarray analysis based on data of 9 lectins giving significant differences (Fold change ≥ 1.5 or ≤0.67). The NFIs of each lectin from this two groups were compared based upon fold-changes.
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