Figure 5: Schematic diagram of the mechanism whereby circRNA_000203 increases cardiac fibrosis.

The binding interaction between circRNA_000203 and miR-26b derepresses the inhibitory effect of miR-26b on Col1a2 and CTGF in cardiac fibroblasts, resulting in the upregulation of Col1a2 and CTGF, and contributing to the enhancement of cardiac fibrosis.