Figure 4: Treatments with r-bursicon homodimers and heterodimer induced AMP production in isolated larval fat body preparations.
From: Relish2 mediates bursicon homodimer-induced prophylactic immunity in the mosquito Aedes aegypti
(A) Accumulation of mRNA encoding five AMP genes following treatments with r-bursicon homodimers or heterodimer. Asterisks indicate significant differences (*P < 0.05; **P < 0.01) from the control group. The growth of E. coli (B) and M. luteus (C) was significantly suppressed by induced larval fat body AMPs. Bars annotated with the same letter are not significantly different, P < 0.05.
